Women with AFLP generally plump mature systemic carnitinedeficiency

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Women with AFLP generally improve soon after delivery, unless the liver damage is severe. As a general rule, AFLP does not usually recur during a subsequent pregnancy. However, AFLP has been found associated in some cases with an abnormality of fatty-acid metabolism. This abnormality is a deficiency of the enzyme long-chain-3-hydroxyacyl-CoA dehydrogenease (LCHAD). The mother (and father) have 50% of normal LCHAD activity and the fetus has no systemic carnitinedeficiency LCHAD activity. The metabolic disease in the baby's liver systemic carnitinedeficiency apparently causes the fatty liver disease in the mother. In systemic carnitinedeficiency cases of AFLP due to LCHAD deficiency, there is a 25% (or greater) risk of AFLP in each pregnancy.
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